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Glant, Tibor
One or more keywords matched the following items that are connected to Glant, Tibor
Item TypeName
Concept Gene Expression Profiling
Concept Gene Expression
Concept Gene Expression Regulation
Academic Article Chemokine gene activation in human bone marrow-derived osteoblasts following exposure to particulate wear debris.
Academic Article Role of fibroblasts and fibroblast-derived growth factors in periprosthetic angiogenesis.
Academic Article The role of fibroblasts and fibroblast-derived factors in periprosthetic osteolysis.
Academic Article Complete coding sequence, deduced primary structure, chromosomal localization, and structural analysis of murine aggrecan.
Academic Article Suppression of osteoblast function by titanium particles.
Academic Article Changes in messenger RNA and protein levels of proteoglycans and link protein in human osteoarthritic cartilage samples.
Academic Article Sex effect on clinical and immunologic quantitative trait loci in a murine model of rheumatoid arthritis.
Academic Article Identification and quantification of disease-related gene clusters.
Academic Article Gene expression profiling in murine autoimmune arthritis during the initiation and progression of joint inflammation.
Academic Article Antigen-induced differential gene expression in lymphocytes and gene expression profile in synovium prior to the onset of arthritis.
Academic Article Interleukin-4 regulates proteoglycan-induced arthritis by specifically suppressing the innate immune response.
Academic Article BALB/c mice genetically susceptible to proteoglycan-induced arthritis and spondylitis show colony-dependent differences in disease penetrance.
Academic Article Excessive bone formation in a mouse model of ankylosing spondylitis is associated with decreases in Wnt pathway inhibitors.
Academic Article Differentially expressed epigenome modifiers, including aurora kinases A and B, in immune cells in rheumatoid arthritis in humans and mouse models.
Academic Article A differential gene expression study: Ptpn6 (SHP-1)-insufficiency leads to neutrophilic dermatosis-like disease (NDLD) in mice.
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  • Gene Expression